TY - JOUR
T1 - Ambulatory ventricular tachycardia
T2 - Characteristics of the initiating beat
AU - Swerdlow, Barry
AU - Axelrod, Paul
AU - Kolman, Benet
AU - Perry, Diane
AU - Mark, Roger
N1 - Funding Information:
From the Department of Medicine and the Thorndike Laboratory, Harvard Medical School and the Beth Israel Hospital; and the Biomedical Engineering Center for Clinical Instrumentation, Massachusetts Institute of Technology. This work was supported in part by Grant HL 07374 from the National Heart, Lung and Blood Institute, United States Public Health Service. Data analysis was performed on the Core Lab computing facilities supported by Grant RR-01032 from the General Clinical Research Centers Program of the Division of Research Resources, National Institutes of Health. Received for publication Dec. 7, 1981; revision received June 17, 1982; accepted July 21, 1982. Reprint requests: Roger G. Mark, M.D., Arrhythmia Israel Hospital, 330 Brookline Ave., Boston, MA 02215.
PY - 1983/12
Y1 - 1983/12
N2 - One hundred ten 24-hour Holter ECG recordings from 82 ambulatory patients with 341 episodes of ventricular tachycardia (VT) were analyzed. Most VT was precipitated by ventricular premature beats (VPBs) with prematurity indices (coupling interval/QT) between 1.0 and 2.0. However, a disproportionate number of VPBs initiating VT occurred early and late in the cardiac cycle compared with isolated VPBs. VT rate was not related to preceding heart rate, but faster basic rates appeared to protect against VT of extended duration. Approximately 80% of episodes of VT were initiated by VPB forms also seen in isolation. Of the 20% of VT initiated by a VPB of new morphology, one third occurred late in the cardiac cycle; almost none were early cycle.
AB - One hundred ten 24-hour Holter ECG recordings from 82 ambulatory patients with 341 episodes of ventricular tachycardia (VT) were analyzed. Most VT was precipitated by ventricular premature beats (VPBs) with prematurity indices (coupling interval/QT) between 1.0 and 2.0. However, a disproportionate number of VPBs initiating VT occurred early and late in the cardiac cycle compared with isolated VPBs. VT rate was not related to preceding heart rate, but faster basic rates appeared to protect against VT of extended duration. Approximately 80% of episodes of VT were initiated by VPB forms also seen in isolation. Of the 20% of VT initiated by a VPB of new morphology, one third occurred late in the cardiac cycle; almost none were early cycle.
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U2 - 10.1016/0002-8703(83)90041-8
DO - 10.1016/0002-8703(83)90041-8
M3 - Article
C2 - 6650354
AN - SCOPUS:0021024370
SN - 0002-8703
VL - 106
SP - 1326
EP - 1331
JO - American heart journal
JF - American heart journal
IS - 6
ER -