No evidence for mutations in a putative subunit of the β-cell ATP-sensitive potassium channel (K-ATP Channel) in Japanese NIDDM patients

Keyphrases

• Diabetes 100%
• Polymorphism 100%
• Family History 100%
• Non-insulin-dependent Diabetes Mellitus (NIDDM) 100%
• KATP Channel 100%
• Cellular ATP 100%
• Mitochondrial Potassium Channels 100%
• Japan 50%
• Missense mutation 50%
• Control Subjects 50%
• Nonsense mutation 50%
• Coding Region 50%
• Pancreatic Cells 50%
• Diabetic 50%
• Single-strand Conformation Polymorphism 50%
• Polymorphism Analysis 50%
• Glucose-stimulated Insulin Secretion 50%
• Genetic Defects 50%
• Allele Frequency 50%
• Cloned Genes 50%
• Japanese Patients 50%
• ATP-sensitive K+ (KATP) Channel 50%

Biochemistry, Genetics and Molecular Biology

• Adenosine Triphosphate 100%
• Adenosine Triphosphate Sensitive Potassium Channel 100%
• Maturity Onset Diabetes of the Young 100%
• Nonsense Mutation 33%
• Missense Mutation 33%
• Genetics 33%
• Coding Region 33%
• Insulin Release 33%
• Potassium Channel 33%
• Gene Frequency 33%
• Glucose 33%

Neuroscience

• Adenosine Triphosphate 100%
• Adenosine Triphosphate Sensitive Potassium Channel 100%
• Missense Mutation 33%
• Potassium Channel 33%
• Nonsense Mutation 33%
• Diabetes Mellitus 33%
• Insulin 33%
• Coding Region 33%
• Allele Frequency 33%
• Genetics 33%
• Glucose 33%