Testing the role of p53 in the expression of genetic instability and apoptosis in ataxia-telangiectasia

Keyphrases

• Apoptosis 100%
• Streptonigrin 100%
• Ataxia Telangiectasia 100%
• Genomic Instability 100%
• T Cells 50%
• Spontaneous Rate 25%
• DNA Damage 25%
• Human Disease 25%
• Signal Transduction Pathway 25%
• Dominant Negative 25%
• Cell Behavior 25%
• Radiation Resistance 25%
• X-ray 25%
• Model Prediction 25%
• Control Cell 25%
• T-lymphoblasts 25%
• Fibroblast Line 25%
• T Gene 25%
• P53 Gene 25%
• Lymphoblast 25%
• Recombination Rate 25%
• Transformant 25%
• Cell Cycle Checkpoint 25%
• Line of Control 25%
• E6 Gene 25%
• DNA Damage-induced Apoptosis 25%
• Stable Transfection 25%
• Mutagens 25%
• HPV18 25%
• Normal Cell Lines 25%
• Human Papillomavirus 25%

Neuroscience

• P53 100%
• Ataxia Telangiectasia 100%
• Programmed Cell Death 100%
• Genetics 100%
• Streptonigrin 80%
• Cell Line 40%
• T Cell 40%
• Lymphoblast 40%
• Cell Function 20%
• Transfection 20%
• Human Papillomavirus 20%
• DNA Damage Response 20%
• DNA Damage 20%
• Stable Transfection 20%
• Mutagen 20%
• Cell Cycle Checkpoint 20%
• Fibroblast 20%
• Signal Transduction 20%

Immunology and Microbiology

• P53 100%
• Ataxia Telangiectasia 100%
• Genomic Instability 100%
• Programmed Cell Death 100%
• Cell Line 40%
• T Cell 40%
• Signal Transduction 20%
• DNA Damage 20%
• X Ray 20%
• Radioresistance 20%
• Genetic Transfection 20%
• Cell Function 20%
• Lymphoblast 20%
• Wart Virus 20%
• Stable Transfection 20%
• DNA Damage Response 20%
• T Lymphoblast 20%
• Human Papillomavirus Type 18 20%
• Cell Cycle Checkpoint 20%
• Fibroblast 20%
• Protein P53 20%