Axon Degeneration: Too Much NMN Is Actually Bad?

Michael S. Cohen

doi:10.1016/j.cub.2017.02.058

Axon Degeneration: Too Much NMN Is Actually Bad?

Michael S. Cohen

Research output: Contribution to journal › Short survey › peer-review

3 Scopus citations

Abstract

The mechanism of axon degeneration is incompletely understood. A recent study demonstrates that transgenic expression of bacterial nicotinamide adenine mononucleotide (NMN) in zebrafish and mice, which decreases NMN levels by converting it to NaMN, protects against axon degeneration.

Original language	English (US)
Pages (from-to)	R310-R312
Journal	Current Biology
Volume	27
Issue number	8
DOIs	https://doi.org/10.1016/j.cub.2017.02.058
State	Published - Apr 24 2017

ASJC Scopus subject areas

General Biochemistry, Genetics and Molecular Biology
General Agricultural and Biological Sciences

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10.1016/j.cub.2017.02.058

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@article{70756453cdb3457abef2e0c147460c77,

title = "Axon Degeneration: Too Much NMN Is Actually Bad?",

abstract = "The mechanism of axon degeneration is incompletely understood. A recent study demonstrates that transgenic expression of bacterial nicotinamide adenine mononucleotide (NMN) in zebrafish and mice, which decreases NMN levels by converting it to NaMN, protects against axon degeneration.",

author = "Cohen, {Michael S.}",

note = "Publisher Copyright: {\textcopyright} 2017 Elsevier Ltd",

year = "2017",

month = apr,

day = "24",

doi = "10.1016/j.cub.2017.02.058",

language = "English (US)",

volume = "27",

pages = "R310--R312",

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TY - JOUR

T1 - Axon Degeneration

T2 - Too Much NMN Is Actually Bad?

AU - Cohen, Michael S.

PY - 2017/4/24

Y1 - 2017/4/24

N2 - The mechanism of axon degeneration is incompletely understood. A recent study demonstrates that transgenic expression of bacterial nicotinamide adenine mononucleotide (NMN) in zebrafish and mice, which decreases NMN levels by converting it to NaMN, protects against axon degeneration.

AB - The mechanism of axon degeneration is incompletely understood. A recent study demonstrates that transgenic expression of bacterial nicotinamide adenine mononucleotide (NMN) in zebrafish and mice, which decreases NMN levels by converting it to NaMN, protects against axon degeneration.

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U2 - 10.1016/j.cub.2017.02.058

DO - 10.1016/j.cub.2017.02.058

M3 - Short survey

C2 - 28441566

AN - SCOPUS:85018796549

SN - 0960-9822

VL - 27

SP - R310-R312

JO - Current Biology

JF - Current Biology

IS - 8

ER -

Axon Degeneration: Too Much NMN Is Actually Bad?

Abstract

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