Effect of nitric oxide synthase inhibition on the cerebral vascular response to hypercapnia in primates

Robert W. McPherson; Jeffrey R. Kirsch; Ramsis F. Ghaly; Richard J. Traystman

doi:10.1161/01.STR.26.4.682

Effect of nitric oxide synthase inhibition on the cerebral vascular response to hypercapnia in primates

Robert W. McPherson, Jeffrey R. Kirsch, Ramsis F. Ghaly, Richard J. Traystman

Research output: Contribution to journal › Article › peer-review

58 Scopus citations

Abstract

Background and Purpose The role of nitric oxide in cerebrovascular response to changes in PCO₂ is unclear. In the present study, we assessed responses at two levels of hypercapnia in a primate model before and after blockade of nitric oxide synthesis.

Original language	English (US)
Pages (from-to)	682-687
Number of pages	6
Journal	Stroke
Volume	26
Issue number	4
DOIs	https://doi.org/10.1161/01.STR.26.4.682
State	Published - Apr 1995
Externally published	Yes

ASJC Scopus subject areas

Clinical Neurology
Cardiology and Cardiovascular Medicine
Advanced and Specialized Nursing

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10.1161/01.STR.26.4.682

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title = "Effect of nitric oxide synthase inhibition on the cerebral vascular response to hypercapnia in primates",

abstract = "Background and Purpose The role of nitric oxide in cerebrovascular response to changes in PCO2 is unclear. In the present study, we assessed responses at two levels of hypercapnia in a primate model before and after blockade of nitric oxide synthesis.",

author = "McPherson, {Robert W.} and Kirsch, {Jeffrey R.} and Ghaly, {Ramsis F.} and Traystman, {Richard J.}",

year = "1995",

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AU - McPherson, Robert W.

AU - Kirsch, Jeffrey R.

AU - Ghaly, Ramsis F.

AU - Traystman, Richard J.

PY - 1995/4

Y1 - 1995/4

N2 - Background and Purpose The role of nitric oxide in cerebrovascular response to changes in PCO2 is unclear. In the present study, we assessed responses at two levels of hypercapnia in a primate model before and after blockade of nitric oxide synthesis.

AB - Background and Purpose The role of nitric oxide in cerebrovascular response to changes in PCO2 is unclear. In the present study, we assessed responses at two levels of hypercapnia in a primate model before and after blockade of nitric oxide synthesis.

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Effect of nitric oxide synthase inhibition on the cerebral vascular response to hypercapnia in primates

Abstract

ASJC Scopus subject areas

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