Abstract
The behavioral and neuroendocrine phenotypes displayed by F2 (129, C57) D2 dopamine receptor-deficient mice (D2R(-/-)) result from a complex interaction between the absence of the receptor and additional polymorphic genetic loci contributed by the parental mouse strains.
Original language | English (US) |
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Pages (from-to) | 373-377 |
Number of pages | 5 |
Journal | Molecular Psychiatry |
Volume | 3 |
Issue number | 5 |
DOIs | |
State | Published - 1998 |
Externally published | Yes |
ASJC Scopus subject areas
- Molecular Biology
- Psychiatry and Mental health
- Cellular and Molecular Neuroscience